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One of the reasons for such a wealth of ways to slow aging in short-lived species is that there are countless possible methods by which researchers can provoke a hormetic response, and the same is true for the other underlying mechanisms that might work to modestly slow aging if manipulated. Everything in cellular biochemistry is connected, so a core mechanism of interest might be tweaked by altering any one of dozens of genes or levels of circulating proteins. Indeed, part of the challenge inherent in this situation is that it is very hard to determine the identity of the core mechanism when there are so many actions that produce benefits, and every change cascades throughout cellular biochemistry. Another thing to bear in mind while reading these sorts of research results is that all of the methods of slowing aging in short-lived animals for which we also have data in humans show that in our species the result on life span is small at best, even when the result on health is worth chasing, as is the case for calorie restriction and exercise:

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